Nature: Cell biology

• 

  Año 2004, Número 2

  • Scaling cell biology: all systems go!

    pág. 79

    

  • The Golgi in plants and animals

    A. Nakano

    pág. 81

    

  • Nuclear transport erupts on the slopes of Mount Etna

    M. A. Powers

    págs. 82-86

    

  • Keeping cartographers busy

    M. S. Hayden

    págs. 87-89

    

  • Modifiying NEMO

    R. T. Hay

    págs. 89-91

    

  • Security measures at the nuclear pore

    A. Schuldt

    pág. 91

    

  • The heat is on with TAC1

    T. Sanchez-Elsner

    págs. 92-93

    

  • Kinetochore sub-structure comes to MIND

    P. Hieter, V. Measday

    págs. 94-95

    

  • Cell of the month: An outer hair cell of the inner ear

    M. Zhang, P. J. Antonelli

    pág. 96

    

  • A physical and functional map of the human TNF-¿/NF-¿B signal transduction pathway

    T. Bouwmeester

    págs. 97-105

    

  • Protein kinase D regulates basolateral membrane protein exit from trans-Golgi network

    C. Yeaman, J. R. Wright, M. I. Ayala

    págs. 106-112

    

  • A novel mechanism for the store-operated calcium influx pathway

    T. Smani, S. I. Zakharov

    págs. 113-120

    

  • ASC is a Bax adaptor and regulates the p53¿Bax mitochondrial apoptosis pathway

    T. Ohtsuka

    págs. 121-128

    

  • The tumour suppressor RASSF1A regulates mitosis by inhibiting the APC¿Cdc20 complex

    J. S. Chang, M. S. Song, S. Song, N. G. Ayad

    págs. 129-137

    

  • The DNA damage checkpoint and PKA pathways converge on APC substrates and Cdc20 to regulate mitotic progression

    J. S. Searle, K. L. Schollaert

    págs. 138-145

    

  • Gadd45ß mediates the NF-¿B suppression of JNK signalling by targeting MKK7/JNKK2

    F. Zazzeroni, S. Papa

    págs. 146-153

    

  • FAK¿Src signalling through paxillin, ERK and MLCK regulates adhesion disassembly

    D. J. Webb, L. A. Whitmore, K. Donais

    págs. 154-161

    

  • Modulation of heat shock gene expression by the TAC1 chromatin-modifying complex

    S. T. Smith

    págs. 162-167

    

  • Senescing human cells and ageing mice accumulate DNA lesions with unrepairable double-strand breaks

    O. A. Sedelnikova

    págs. 168-170